Proliferating cells use glutamine in excess of other amino acids to produce a variety of cellular components - proteins, nucleic acids, glycosyl chains to name a few. Because of their high biosynthetic burden and poor vascularization, solid tumors tend to have lower levels of glutamine in their microenvironment. Our goal is to understand how various cell types within the tumor microenvironment sense, adapt and respond to shortages of this critical nutrient.
What biochemical, positional and signaling requirements must be met in order to unlock the cells' ability to make glutamine de novo for themselves and their neighbors?
Some transcriptional regulators (and other proteins) contain long polyglutamine tracts within their primary sequence. Can these mysterious elements act as built-in sensors of glutamine supply to help cells tailor their decisions to nutrient availability?
We are building and validating new tools for profiling amino acid depletion at a single-cell resolution within tumors and normal tissue contexts.
Alata
Alice
Open Sans
Noto Sans
Bebas Neue
Great Vibes
Rock Salt
Exo
Belgrano
Overlock
Cinzel
Indie Flower
Staatliches
Roboto Slab
Lato
Noto Serif
Open Sans
Montserrat
Ubuntu
Rubik
Delius
Amiri
Montserrat